Thumbnail Image

Chaudhary Charan Singh Haryana Agricultural University, Hisar

Chaudhary Charan Singh Haryana Agricultural University popularly known as HAU, is one of Asia's biggest agricultural universities, located at Hisar in the Indian state of Haryana. It is named after India's seventh Prime Minister, Chaudhary Charan Singh. It is a leader in agricultural research in India and contributed significantly to Green Revolution and White Revolution in India in the 1960s and 70s. It has a very large campus and has several research centres throughout the state. It won the Indian Council of Agricultural Research's Award for the Best Institute in 1997. HAU was initially a campus of Punjab Agricultural University, Ludhiana. After the formation of Haryana in 1966, it became an autonomous institution on February 2, 1970 through a Presidential Ordinance, later ratified as Haryana and Punjab Agricultural Universities Act, 1970, passed by the Lok Sabha on March 29, 1970. A. L. Fletcher, the first Vice-Chancellor of the university, was instrumental in its initial growth.

Browse

20201
Reset filters
Subject: Genetics and Plant Breeding × Author: Chaudhary, Mohit × End date: 2023 × Start date: 2020 × Type: Thesis × Thesis Type: Ph.D ×
Reset filters

Search Results

Now showing 1 - 1 of 1
  • Thumbnail Image
    ThesisItemOpen Access
    Genetic architecture of yield traits and molecular characterization of important quality characters in Indian Mustard [Brassica juncea (L.) Czern & Coss. .
    (CCSHAU,HiSAR, 2020) Chaudhary, Mohit; Ram Avtar
    The present investigation was carried out to study “Genetic architecture of yield traits and molecular characterization of important quality characters in Indian mustard (Brassica juncea L. Czern & Coss.)”. Six generations each i.e. parents, F1, F2, BC1 and BC2 were generated from two crosses (RH 0749 x RLC-3 and RH 0749 x PDZ-1) using three varieties (RH 0749, RLC-3 and PDZ-1) having extreme level of erucic acid and glucosinolates content, during rabi 2015-16 and rabi 2016-17. Observations were recorded on following characters viz., days to maturity, plant height (cm), number of primary branches, number of secondary branches, main shoot length (cm), number of siliqua on main shoot, siliqua length (cm), number of seed per siliqua, 1000-seed wt. (g), seed yield per plant (g), oil content (%), oleic acid (%), linoleic acid(%), linolenic acid (%), erucic acid (%) and glucosinolates content (μ mol/g defatted meal). Generation mean analysis revealed significant differences for most of the trait studied. Individual scaling test as well as χ2 in joint scaling test were observed significant for most of the characters studied. Hence, six parameter model was suggested. In general, additive gene effects were found to play significant role for 1000-seed weight, seed yield per plant and few other associated characters. For quality traits, additive gene effects were more important than dominant gene effects. Duplicate epistasis were observed for most of the characters studied in both the crosses. Inheritance of erucic acid and glucosinolates content were also studied. For erucic acid content no significant differences were observed between F1s and reciprocals in both the crosses. The classes of erucic acid in F2 generation fits well in segregation ration of 1:4:6:4:1 indicated digenic inheritance of erucic acid with additive effect. Minimum number of effective factor pairs for erucic acid were ranged from 1.77 to 1.84 indicated two gene control of this trait. Total glucosinolates content were found to influenced by maternal/ cytoplasm effect. Minimum number of effective factor pair were ranged from 3.87 to 4.45 in two crosses indicated that inheritance of total glucosinolates content were under control of at least 4 to 5 gene pairs. The PCR amplification of primers related to FAE1 gene revealed a single band. Similarly, amplification of primers related to glucosinolates content also revealed monomorphic band between contrasting parents. This suggested that variation between parents were due to minor or point mutation with the possibilities of SNPs being responsible for the variation.