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Anand Agricultural University, Anand

Anand Agricultural University (AAU) was established in 2004 at Anand with the support of the Government of Gujarat, Act No.(Guj 5 of 2004) dated April 29, 2004. Caved out of the erstwhile Gujarat Agricultural University (GAU), the dream institution of Sardar Vallabhbhai Patel and Dr. K. M. Munshi, the AAU was set up to provide support to the farming community in three facets namely education, research and extension activities in Agriculture, Horticulture Engineering, product Processing and Home Science. At present there seven Colleges, seventeen Research Centers and six Extension Education Institute working in nine districts of Gujarat namely Ahmedabad, Anand, Dahod, Kheda, Panchmahal, Vadodara, Mahisagar, Botad and Chhotaudepur AAU's activities have expanded to span newer commodity sectors such as soil health card, bio-diesel, medicinal plants apart from the mandatory ones like rice, maize, tobacco, vegetable crops, fruit crops, forage crops, animal breeding, nutrition and dairy products etc. the core of AAU's operating philosophy however, continues to create the partnership between the rural people and committed academic as the basic for sustainable rural development. In pursuing its various programmes AAU's overall mission is to promote sustainable growth and economic independence in rural society. AAU aims to do this through education, research and extension education. Thus, AAU works towards the empowerment of the farmers.

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  • ThesisItemOpen Access
    STUDIES ON PROTECTIVE EFFECT OF DIMETHYL SULFOXIDE (DMSO) IN EXPERIMENTALLY INDUCED INTESTINAL ISCHEMIA IN CALVES
    (AAU, Anand, 1993) Garara, Shailesh V.; Jani, B. M.
    The experiment was conducted on twelve 6 to 12 months old cow calves to judge the nature and extent of reperfusion injury after intestinal ischemia and to evaluate protective effect of dimethyl sulfoxide (DMSO), All the animals were subjected to one complete and one incomplete ischemic strangulation obstruction (ISO) of about 25 cm jejunal segments for the duration of 6 hours. After 6 hours of ischemia, reperfusion of the intestinal segments was established by releasing the vascular obstructions. The animals were randomly divided into two equal groups (Group A and B), The animals of Group A served as control ones and no treatment was carried out at the time of reperfusion, while animals of Group B were given 20 per cent DMSO solution in normal saline intravenously at the rate of 2 g/kg body weight at the time of reperfusion. The protective effect of DMSO was ascertained by evaluating standard clinical criteria, fluorescent pattern and histopathological findings at 24 hours reperfusion period. Physical changes like malaise, dullness, depression, change in rectal temperature, respiration rate and heart rate were of no significance either in ascertaining ischemic damage or in evaluating the protective effect of DMSO. Grossly, serosa of ischemic segments in control group revealed severe degenerative changes including moderate to severe adhesions, oedema and congestion, and reddish colouration while that of treated segments showed absence of adhesion, pinkish colour, hyperemia and slight oedema and congestion. Similarly mucosal changes were severe in control segments and were of very mild degree in treated segments. Following fluorescein dye injection, the treated segments showed viable fluorescent pattern while that of control segments showed non-viable fluorescent pattern. Histopathologically, control segments showed stunted mucosal villi lined by low columnar to cuboidal epithelial cells with few villi naked. The lumen contained desquamated epithelial cells with haemorrhages. Mucosa of treated segments was almost normal. Degenerative changes in submucosa, lamina propria and serosa were severe in control group while these clianges were either absent or were of very mild degree in treated segments. Based on SCC, fluorescein dye technique, and histopathological observations, it was-concluded that the intravenous DMSO therapy at the time of reperfusion could be a safe and effective supplementary therapy to combat ischemic intestinal disorders in cattle.